youtu.be — Nice way to spend some time with your kids, and get a workout! Fun, fitness, fruits, come join us at the raw fruit festival in Spain, 12 to 16 of September 2012. Stay young! More infos and online registration on http://festival.811-friendly.net

youtube.com — Picking oranges on our farm.For real fruits, really ripe, straight from the farm, 5 days of fun with great people, in nature, come to the 811-friendly raw fruit festival in Spain, September 12-16 2012http://festival.811-friendly.net

veganhealth.org — Iodine Status of Vegetarians Iodine is only found inconsistently in plant foods, depending on the iodine content of the soil. Food grown near the ocean tends to be higher in iodine. Iodine is consistently found in only a few foods such as dairy products (iodine solutions are used to clean the cows' teats and dairy equipment and end up in the milk) and seafood (including seaweed). In a 2011 cross-sectional study from the Boston area, urinary iodine levels of 78 lacto-ovo vegetarians and 62 vegans were measured. People with previously diagnosed thyroid problems were excluded from the study. According to the authors, "Population iodine sufficiency is defined by median urinary iodine concentrations 100 µg/l or greater in adults and 150 µg/l or greater in pregnancy." Median urinary iodine concentration of vegans (79 µg/l; range 7 – 965 µg/l) was significantly lower than vegetarians (147 µg/l; range 9 – 779 µg/l). Markers of thyroid function were similar in both groups and in the normal range; one vegan and no vegetarians had abnormal thyroid function. Most of the vegans were making no effort to insure adequate iodine intake. Iodine deficiency is not as much of a problem for U.S. vegans as it is for European vegans, whose food supply contains less iodine. Studies have shown that vegans in Europe (where salt is minimally iodized) who do not supplement (as well as those who oversupplement) have indications of abnormal thyroid function.

scienceblogs.com — In most texts and sources that I've read, the germ theory of disease is stated something like, "Many diseases are caused by microorganisms." We could argue whether viruses count as microorganisms, but for purposes of the germ theory they do. (Most biologists do not consider viruses to be true living organisms, because they consist of nothing more than genetic material wrapped in a protein coat and lack the ability to reproduce without infecting the cell of an organism.) Now, let's take a look at the latest germ theory denialist idiocy I've come across. The first one, not surprisingly, I found on NaturalNews.com. Surprisingly, it was not written by Mike Adams, but rather by someone named Paul Fassa, who proclaims You have been lied to about germs. It should have been called "You are about to be lied to about germs." First, though, since this article wasn't by the usual science-hating loon Mike Adams, I was curious just who Paul Fassa is. I had never heard of him before. It didn't take long to find Fassa's Twitter account and then from there his blog Health Maven, which bills itself as an "escape from the medical mafia matrix." Interesting. Why does it appear that any time I come across a germ theory denialist like Fassa, he's someone who uses terms like "medical mafia matrix"? I don't know, but such people also tend to write introductory paragraphs like this: We have been taught to fear germs, pathogens, viruses, and bacteria that invade us from out there. This is the Pasteur model of disease contagion. This creates a dependency on Big Pharma to protect us from invading microbes, each having one form (monomorphic) and creating one specific disease. Pasteur`s model of disease won over rival Claude Bernard`s more accurate argument of the inner terrain. Pasteur`s declaration, though serving the coffers of Big Pharma, creates more questions: How come some get a disease that`s going around and others don`t? How do all these new bugs come out of nowhere to haunt us? Why do vaccines and antibiotics ultimately fail and create super bugs? These questions are answered by understanding the inner terrain and pleomorphism. Note how Fassa first misrepresents the Pasteur model of disease. This is common among germ theory denialists, in my experience. They tend to assume that germ theory states that pathogenic microbes are 100% infectious and always cause disease. Consequently, when people are exposed to pathogenic microbes and don't become ill, people like Fassa point to that as evidence that germ theory is invalid. After all, the germ didn't cause disease, at least in this one case! That must mean that all of germ theory is wrong! Concrete thinking, thy name is Fassa (and other germ theory denialists.) It's rather odd that even most teenagers can understand that catching an infectious disease is dependent not just on the microbe but each person's resistance to that microbe. This is the same thing that mystifies HIV/AIDS denialists, who seem to view the observation that most exposures to HIV do not result in AIDS as some sort of devastating indictment of the hypothesis that HIV causes AIDS. Add to that a long asymptomatic period and highly variable rates of progression, and HIV/AIDS denialists, who are--let's face it--really nothing more than a subtype of germ theory denialists who deny vehemently that one particular germ causes disease have all the doubt they need. But I digress. Also notice Fassa's early and immediate invocation of the pharma shill gambit. If there's another thing about germ theory denialism, it's that those who cling to it tend to be extremely distrustful of big pharma. I realize that in many cases big pharma deserves a lot of mistrust; its record in many areas demands it. What distinguishes many of these germ theory denialists is that they take healthy skepticism and take it to a pathological extreme. They also seem to think that the reason that antibiotics ultimately fail is because germ theory is invalid, which reveals an incredible ignorance of how antibiotics work. Helloooo! Evolution? Ever heard of it? Bacteria are incredibly good at evolving under the selective pressure of antibiotics. That's what creates superbugs, that and our tendency to overuse antibiotics. But what is the "inner terrain" and pleomorphism? This is where we find the "intellectual" basis of rejection of germ theory. As is the case with many alt-med beliefs, this basis harkens back to "ancient" knowledge (or at least 150 year old knowledge). It harkens back to Antoine Béchamp, who did indeed postulate nearly the exact opposite of what Pasteur did: that microorganisms were not the cause of disease but rather the consequence of disease, that injured or diseased tissues produced them and that it was the health of the organism that mattered, not the microorganisms. Basically, Béchamp's idea, known as the pleomorphic theory of disease, stated that bacteria change form (i.e., demonstrate pleomorphism) in response to disease, not as a cause of disease. In other words, they arise from tissues during disease states; they do not invade from the external world. Béchamp further proposed that bacteria arose from structures that he called microzymas, which to him referred to a class of enzymes. Béchamp postulated that microzymas are normally present in tissues and that their effects depended upon the cellular terrain. Ultimately, Pasteur's theory won out over that of Béchamp, based on evidence, but Béchamp was influential at the time. Given the science and technology of the time, Béchamp's hypothesis was not entirely unreasonable. It was, however, superseded by Pasteur's germ theory of disease and Koch's later work that resulted in Koch's postulates. What needs to be remembered is that not only did Béchamp's hypothesis fail to be confirmed by scientific evidence, but his idea lacked the explanatory and predictive power of Pasteur's theory. Fassa is sort of correct about one thing, though. Béchamp's idea was basically something like this: The inner terrain includes our immune system, organ tissues, and blood cells. Those who stepped out of line from Pasteur`s dogma asserted that the inner terrain was more vital for remaining disease free than searching for new antibiotics and vaccines to kill bacteria and viruses. As an analogy, flies don`t create garbage. But garbage attracts flies that breed maggots to create even more flies. Removing garbage is more effective than spraying toxic chemicals, which endanger human and animal life, around the house. Similarly, adding toxins to humans is not as effective as cleaning out the inner terrain. As I said, there's a grain of truth there, namely that the condition of the body and a person's immune system does matter. Specifically, it is true that the condition of the "terrain" (the body) does matter when it comes to infectious disease. Debilitated people do not resist the invasion of microorganisms as well as strong, healthy people. Of course, another thing to remember is that the "terrain" can facilitate the harmful effect of microorganisms in unexpected ways. For example, certain strains of the flu (as in 1918 and H1N1) are more virulent in the young because the young mount a more vigorous immune response. However, latter day Béchamp worshipers fetishize this idea to the point of claiming that the "inner terrain" is all that matters and that bacteria and viruses are manifestations, not causes, of disease. It goes beyond that, though. According to Béchamp, it's said: Blood is alive. It is not a liquid, but a mobile tissue (Béchamp was the first to describe blood thus). The things in our blood are alive. And one thing modern medicine does not accept is that something like a bacterium can change into a yeast that can turn into a fungus that can turn into a mold. We've talked about this in previous newsletters; it is called pleomorphism. Pleo meaning many and morph meaning form or body. This is, of course, complete nonsense. Bacteria cannot change into yeast or vice-versa, while yeasts are organisms in the kingdom Fungi. Dimorphic fungi can exist as a mold/hyphal/filamentous form or as yeast, but this fact does not invalidate the germ theory of disease. Indeed, some of these fungi are pathogens, such as Blastomyces dermatitidis, Histoplasma capsulatum, and Sporothrix schenckii. The misunderstanding of microbiology required to accept the rejection of germ theory in favor of Béchamp's ideas is staggering. Yet they remain very influential. Not among scientists, of course. Science moved on a long time ago. Rather, they remain influential among cranks. By Orac

scienceblogs.com — In fact, most of the bacteria, fungi, and yeast that inhabit our bodies are either beneficial or neutral; our digestive systems wouldn't function properly without the bacteria that live in our colons. They're responsible for breaking down some polysaccharides, starches, fibers into forms that can be absorbed. Without these wee beasties, there would be a lot of the components of our food that we'd have a hard time absorbing. They even produce some vitamins and facilitate their absorption. But that's not all. The normal gut flora play a major role in preventing infection by crowding out pathogenic bacteria. That's one reason why antibiotics can result in severe diarrheal diseases. They kill off the "good bacteria," leaving the "bad bacteria" to proliferate. They can also stimulate the development of the gut's mucosal immune system. Truly, it is a symbiotic relationship between us humans and our bacterial flora, which colonize our guts shortly after we are born and stay with us for the rest of our lives. by Orac

nature.com — Modification of immune responses to exercise by carbohydrate, glutamine and anti-oxidant supplements Immunosuppression in athletes involved in heavy training is undoubtedly multifactorial in origin. Training and competitive surroundings may increase the athlete's exposure to pathogens and provide optimal conditions for pathogen transmission. Heavy prolonged exertion is associated with numerous hormonal and biochemical changes, many of which potentially have detrimental effects on immune function. Furthermore, improper nutrition can compound the negative influence of heavy exertion on immunocompetence. An athlete exercising in a carbohydrate-depleted state experiences larger increases in circulating stress hormones and a greater perturbation of several immune function indices. The poor nutritional status of some athletes may predispose them to immunosuppression. For example, dietary deficiencies of protein and specific micronutrients have long been associated with immune dysfunction. Although it is impossible to counter the effects of all of the factors that contribute to exercise-induced immunosuppression, it has been shown to be possible to minimize the effects of many factors. Athletes can help themselves by eating a well-balanced diet that includes adequate protein and carbohydrate, sufficient to meet their energy requirements. This will ensure a more than adequate intake of trace elements without the need for special supplements. Consuming carbohydrate (but not glutamine or other amino acids) during exercise attenuates rises in stress hormones, such as cortisol, and appears to limit the degree of exercise-induced immunosuppression, at least for non-fatiguing bouts of exercise. Evidence that high doses of anti-oxidant vitamins can prevent exercise-induced immunosuppression is also lacking.

ds9.botanik.uni-bonn.de —  ROOT APEX – THE ANTERIOR POLE OF PLANT BODY Root Apices represent the Anterior Pole: Specialized for uptake of nutrients and for neuronal activities. Importantly, new roots are formed endogenously (recapitulation of embryogenesis). Shoot Apices represent the Posterior Pole: Specialized for photosynthesis (which is dispensable in holoparasitic plants like Rafflesia) and for sexual reproduction. The flower is the perfect form of the shoot. Shoots harbor plant organs of excretion, trichomes and hydathodes. Moreover, stomata allow gas exchange. Similarly as sexual organs, also organs of plant excretion and stomata are located at the posterior part of the plant body. Even more, hydathodes seem to function in analogy to kidney (Pilot et al. 2004, Plant Cell 16: 1827-1840). Roots are essential whereas shoots are dispensable: In holoparasitic plants, such as Rafflesia, roots are transformed into haustoria while the green part of the plant is missing completely. Nevertheless, haustoria of Rafflesia form the largest flowers in the plant kingdom which reveals that this unique organism really belongs to plants. AUXIN – PLANT NEUROTRANSMITTER Auxin: Represents a plant-specific neurotransmitter and is transported, in a light- and gravity-dependent manner, preferentially along the anterior-posterior axis of the plant body. Auxin induces the formation of both vascular strands (plant nerves) and roots (which harbour the “serial plant brain”). Roots and Auxin: Root apices represent major sinks for the polar auxin transport. Root apices are extremely sensitive towards externally applied auxin, and lateral root formation is induced by this means. Moreover, auxin rapidly regulates vesicle trafficking and gene expression in roots. Initiation of lateral root primordia is an endogenous process resembling early embryogenesis. In contrast, new shoots and leaves are formed exogenously. CELLULAR END-POLES – PLANT SYNAPSES Plant Synapses: Stable actin-supported adhesive domains (known as end-poles or cross-walls) between adjacent plant cells across which auxin and other chemical signals are transported via actin-based vesicular trafficking pathways. Besides these developmental plant synapses, plants are also capable of forming cell-to-cell junctions with cells of another organisms (plants – fungi – bacteria) corresponding to what is defined as an ‘immunological synapse’. These specialized cell-to-cell adhesion domains involve the plasma membranes of two different organisms opposing each other. Such adhesive domains are also sites of active cell-to-cell transport of molecules and metabolites. VASCULAR STRANDS – PLANT NERVES Vascular Strands: The basic units of the vascular system represent both plant nerves as well as a plant endoskeleton. Leaves contain single strands which combine to form the vascular bundles of the stem, and the vascular cylinder of the root. In roots, the largest portion of the organ is the vascular tissue, and its strands (plant nerves) are supported by numerous cells forming the vascular cylinder. Phloem: Supracellular axon-like ‘channel' interconnecting shoot and root apices. Phloem is specialized for transmission of action-potential-driven electric signals. Axon-like means that it is specialized for the rapid transfer of RNA molecules but does not accomplish ribosome assembly and mRNA translation. Xylem: Non-living and water-filled tubes specialized for transmission of hydraulic signals which are self-transmitting waves induced and driven by changes in hydrostatic pressure. ROOT APICES INTERCONNECTED VIA VASCULAR CYLINDERS – SERIAL NERVOUS SYSTEM OF PLANT Plant Brain: Each root apex harbours a unit of nervous system of plants. The number of root apices in the plant body is high and all brain-units are interconnected via vascular strands (plant nerves) with their polarly-transported auxin (plant neurotransmitter), to form a serial (parallel) nervous system of plants. The computational and informational capacity of this nervous system based on interconnected parallel units is predicted to be higher than that of the diffuse nervous system of lower animals, or the central nervous system of higher animals/humans.

5e.plantphys.net —  A Companion to Plant Physiology, Fifth Edition by Lincoln Taiz and Eduardo Zeiger Topics 1. Plant Cells Topic 1.1, Model Organisms Topic 1.2, The Plant Kingdom Topic 1.3, Flower Structure and the Angiosperm Life Cycle Topic 1.4, Plant Tissue Systems: Dermal, Ground, and Vascular Topic 1.5, The Structures of Chloroplast Glycosylglycerides Topic 1.6, A Model for the Structure of Nuclear Pores Topic 1.7, The Proteins Involved in Nuclear Import and Export Topic 1.8, Protein Signals Used to Sort Proteins to their Destinations Topic 1.9, SNAREs, Rabs, and Coat Proteins Mediate Vesicle Formation, Fission, and Fusion Topic 1.10, ER Exit Sites (ERES) and Golgi Bodies Are Interconnected Topic 1.11, Specialized Vacuoles in Plant Cells Topic 1.12, Actin-Binding Proteins Regulate Microfilament Growth Topic 1.13, Kinesins Are Associated with Other Microtubules and Chromatin Topic 1.14, Chapter One References 2. Genome Organization and Gene Expression Topic 2.1, Recombination Mapping and Gene Cloning Topic 2.2, Transposon Tagging 3. Water and Plant Cells Topic 3.1, Calculating Capillary Rise Topic 3.2, Calculating Half-Times of Diffusion Topic 3.3, Alternative Conventions for Components of Water Potential Topic 3.4, Temperature and Water Potential Topic 3.5, Can Negative Turgor Pressures Exist in Living Cells? Topic 3.6, Measuring Water Potential Topic 3.7, The Matric Potential Topic 3.8, Wilting and Plasmolysis Topic 3.9, Understanding Hydraulic Conductivity Topic 3.10, Chapter Three References 4. Water Balance of Plants Topic 4.1, Irrigation Topic 4.2, Physical Properties of Soils Topic 4.3, Calculating Velocities of Water Movement in the Xylem and in Living Cells Topic 4.4, Leaf Transpiration and Water Vapor Gradients Topic 4.5, Chapter Four References 5. Mineral Nutrition Topic 5.1, Symptoms of Deficiency in Essential Minerals - Wade Berry, UCLA Topic 5.2, Observing Roots below Ground Topic 5.3, Chapter Five References 6. Solute Transport Topic 6.1, Relating the Membrane Potential to the Distribution of Several Ions across the Membrane: The Goldman Equation Topic 6.2, Patch Clamp Studies in Plant Cells Topic 6.3, Chemiosmosis in Action Topic 6.4, Kinetic Analysis of Multiple Transporter Systems Topic 6.5, ABC Transporters in Plants Topic 6.6, Transport Studies with Isolated Vacuoles and Membrane Vesicles Topic 6.7, Chapter Six References 7. Photosynthesis: The Light Reactions Topic 7.1, Principles of Spectrophotometry Topic 7.2, The Distribution of Chlorophylls and Other Photosynthetic Pigments Topic 7.3, Quantum Yield Topic 7.4, Antagonistic Effects of Light on Cytochrome Oxidation Topic 7.5, Structures of Two Bacterial Reaction Centers Topic 7.6, Midpoint Potentials and Redox Reactions Topic 7.7, Oxygen Evolution Topic 7.8, Photosystem I Topic 7.9, ATP Synthase Topic 7.10, Mode of Action of Some Herbicides Topic 7.11, Chlorophyll Biosynthesis Topic 7.12, Chapter Seven References 8. Photosynthesis: The Carbon Reactions Topic 8.1, CO2 Pumps Topic 8.2, How the Calvin–Benson Cycle Was Elucidated Topic 8.3, Rubisco: A Model Enzyme for Studying Structure and Function Topic 8.4, Energy Demands for Photosynthesis in Land Plants Topic 8.5, Rubisco Activase Topic 8.6, Thioredoxins Topic 8.7, Operation of the C2 Oxidative Photosynthetic Carbon Cycle Topic 8.8, Carbon Dioxide: Some Important Physicochemical Properties Topic 8.9, Three Variations of C4 Metabolism Topic 8.10, Single-Cell C4 Photosynthesis Topic 8.11, Photorespiration in CAM plants Topic 8.12, Glossary of Carbohydrate Biochemistry Topic 8.13, Starch Architecture Topic 8.14, Fructans Topic 8.15, Chloroplast Phosphate Translocators Topic 8.16, Chapter Eight References 9. Photosynthesis: Physiological and Ecological Considerations Topic 9.1, Working with Light Topic 9.2, Heat Dissipation from Leaves: The Bowen Ratio Topic 9.3, The Geographic Distributions of C3 and C4 Plants Topic 9.4, Calculating Important Parameters in Leaf Gas Exchange Topic 9.5, Prehistoric Changes in Atmospheric CO2 Topic 9.6, Projected Future Increases in Atmospheric CO2 Topic 9.7, Using Carbon Isotopes to Detect Adulteration in Foods Topic 9.8, Reconstruction of the Expansion of C4 Taxa Topic 9.9, Chapter Nine References 10. Translocation in the Phloem Topic 10.1, Sieve Elements as the Transport Cells between Sources and Sinks - Susan Dunford, University of Cincinnati Topic 10.2, An Additional Mechanism for Blocking Wounded Sieve Elements in the Legume Family - Susan Dunford, University of Cincinnati Topic 10.3, Sampling Phloem Sap - Susan Dunford, University of Cincinnati Topic 10.4, Nitrogen Transport in the Phloem - Susan Dunford, University of Cincinnati Topic 10.5, Monitoring Traffic on the Sugar Freeway: Sugar Transport Rates in the Phloem - Susan Dunford, University of Cincinnati Topic 10.6, Alternative Views of Pressure Gradient in Sieve Elements: Large or Small Gradients? - Susan Dunford, University of Cincinnati Topic 10.7, Experiments on Phloem Loading - Susan Dunford, University of Cincinnati Topic 10.8, Experiments on Phloem Unloading - Susan Dunford, University of Cincinnati Topic 10.9, Allocation in Source Leaves: The Balance between Starch and Sucrose Synthesis - Susan Dunford, University of Cincinnati Topic 10.10, Partitioning: The Role of Sucrose-Metabolizing Enzymes in Sinks Topic 10.11, Possible Mechanisms Linking Sink Demand and Photosynthetic Rate in Starch Storers - Susan Dunford, University of Cincinnati Topic 10.12, Proteins and RNAs: Signal Molecules in the Phloem Topic 10.13, Chapter Ten References - Susan Dunford, University of Cincinnati 11. Respiration and Lipid Metabolism Topic 11.1, Isolation of Mitochondria - Ian M. Møller, Aarhus University, Denmark; Allan G. Rasmusson, Lund University, Sweden Topic 11.2, The Q-Cycle Explains How Complex III Pumps Protons across the Inner Mitochondrial Membrane - Allan G. Rasmusson, Lund University, Sweden; Ian M. Møller, Aarhus University, Denmark Topic 11.3, Multiple Energy Conservation Bypasses in Oxidative Phosphorylation of Plant Mitochondria - Allan G. Rasmusson, Lund University, Sweden; Ian M. Møller, Aarhus University, Denmark Topic 11.4, FoF1-ATP Synthases: The World′s Smallest Rotary Motors - Lincoln Taiz, University of California, Santa Cruz, California, USA Topic 11.5, Transport Into and Out of Plant Mitochondria - Allan G. Rasmusson, Lund University, Sweden; Ian M. Møller, Aarhus University, Denmark Topic 11.6, The Genetic System in Plant Mitochondria Has Several Special Features - Allan G. Rasmusson, Lund University, Sweden; Ian M. Møller, Aarhus University, Denmark Topic 11.7, Does Respiration Reduce Crop Yields? - James N. Siedow, Duke University, North Carolina, USA; Ian M. Møller, Aarhus University, Denmark; Allan G. Rasmusson, Lund University, Sweden Topic 11.8, The Lipid Composition of Membranes Affects the Cell Biology and Physiology of Plants - John Browse, Washington State University Topic 11.9, Utilization of Oil Reserves in Cotyledons - John Browse, Washington State University Topic 11.10, Chapter 11 References 12. Assimilation of Mineral Nutrients Topic 12.1, Development of a Root Nodule Topic 12.2, Measurement of Nitrogen Fixation Topic 12.3, The Synthesis of Methionine Topic 12.4, Oxygenases Topic 12.5, Chapter Twelve References 13. Secondary Metabolites and Plant Defense Topic 13.1, Cutin, Waxes, and Suberin Topic 13.2, Structure of Various Triterpenes Topic 13.3, The Shikimic Acid Pathway Topic 13.4, Detailed Chemical Structure of a Portion of a Lignin Molecule Topic 13.5, Chapter Thirteen References 15. Cell Walls: Structure, Biogenesis, and Expansion Topic 15.1, Plant Cell Walls Play a Major Role in Carbon Flow through Ecosystems Topic 15.2, Terminology for Polysaccharide Chemistry Topic 15.3, Molecular Model for the Synthesis of Cellulose and Other Wall Polysaccharides That Consist of a Disaccharide Repeat Topic 15.4, Matrix Components of the Cell Wall Topic 15.5, The Mechanical Properties of Cell Walls: Studies With Nitella Topic 15.6, Wall Degradation and Plant Defense Topic 15.7, Structure of Biologically Active Oligosaccharins Topic 15.8, Glucanases and Other Hydrolytic Enzymes May Modify the Matrix Topic 15.9, Chapter Fifteen References 16. Growth and Development Topic 16.1, Embryonic Dormancy Topic 16.2, Rice Embryogenesis Topic 16.3, Polarity of Fucus Zygotes Topic 16.4, Azolla Root Development Topic 16.5, Class III HD-Zip Transcription Factors Promote Adaxial Development through a microRNA-Sensitive Mechanism Topic 16.6, During Senescence Photoactive Chlorophyllide Is Converted into a Colorless Chlorophyll Catabolite Topic 16.7, Chapter Sixteen References 17. Phytochrome and Light Control of Plant Development Topic 17.1, Mougeotia: A Chloroplast with a Twist Topic 17.2, Phytochrome and High-Irradiance Responses Topic 17.3, The Origins of Phytochrome as a Bacterial Two-Component Receptor Topic 17.4, Profiling Gene Expression in Plants Topic 17.5, Two-Hybrid Screens and Co-immunoprecipitation Topic 17.6, Phytochrome Effects on Ion Fluxes Topic 17.7, Microarray Analysis of Shade Avoidance Topic 17.8, Chapter Seventeen References 18. Blue-Light Responses: Morphogenesis and Stomatal Movements Topic 18.1, Blue-Light Sensing and Light Gradients Topic 18.2, Guard Cell Osmoregulation and a Blue Light-Activated Metabolic Switch Topic 18.3, The Coleoptile Chloroplast Topic 18.4, Phytochrome-Mediated Responses in Stomata Topic 18.5, Chapter Eighteen References 20. Gibberellins: Regulators of Plant Height and Seed Germination Topic 20.1, Structures of Some Important Gibberellins and Their Precursors, Derivatives, and Inhibitors of Gibberellin Biosynthesis - Valerie Sponsel, Biology Department, University of Texas, San Antonio, Texas, USA Topic 20.2, Commercial Uses of Gibberellins - Valerie Sponsel, Biology Department, University of Texas, San Antonio, TX, USA Topic 20.3, Gibberellin Biosynthesis - Valerie Sponsel, Biology Department, University of Texas, San Antonio, TX, USA Topic 20.4, Gas Chromatography—Mass Spectrometry of Gibberellins - Valerie Sponsel, Biology Department, University of Texas, San Antonio, TX, USA Topic 20.5, Environmental Control of Gibberellin Biosynthesis - Valerie Sponsel, Biology Department, University of Texas, San Antonio, TX, USA Topic 20.6, Auxin Can Regulate Gibberellin Biosynthesis - Jocelyn A. Ozga and Dennis M. Reinecke, Plant BioSystems Group, Department of Agricultural, Food, and Nutritional Science, University of Alberta, Edmonton, Alberta, Canada T6G 2P5 Topic 20.7, Negative Regulators of GA Response - Valerie Sponsel, Biology Department, University of Texas, San Antonio, TX, USA Topic 20.8, Effects of GAs on Flowering - Valerie Sponsel, Biology Department, University of Texas, San Antonio, TX, USA Topic 20.9, DELLA Proteins as Integrators of Multiple Signals - Stephen G. Thomas, Rothamsted Research, Harpenden, United Kingdom Topic 20.10, Chapter Twenty References 21. Cytokinins: Regulators of Cell Division Topic 21.1, Cultured Cells Can Acquire the Ability to Synthesize Cytokinins Topic 21.2, Structures of Some Naturally Occurring Cytokinins Topic 21.3, Various Methods Are Used to Detect and Identify Cytokinins Topic 21.4, The Biologically Active Form of Cytokinin Is the Free Base Topic 21.5, Cytokinins Are Also Present in Some tRNAs in Animal and Plant Cells Topic 21.6, The Structures of Opines Topic 21.7, The Ti Plasmid and Plant Genetic Engineering Topic 21.8, Phylogenetic Tree of IPT genes Topic 21.9, A Root-Derived Hormone, Strigolactone, Is Involved in the Suppression of Branching in Shoots Topic 21.10, Cytokinin Can Promote Light-Mediated Development Topic 21.11, Cytokinins Promote Cell Expansion and Greening in Cotyledons Topic 21.12, Cytokinins Interact with Elements of the Circadian Clock Topic 21.13, Chapter Twenty-One References 22. Ethylene: The Gaseous Hormone Topic 22.1, Ethylene in the Environment Arises Biotically and Abiotically Topic 22.2, Ethylene Readily Undergoes Oxidation Topic 22.3, Ethylene Can Be Measured by Gas Chromatography Topic 22.4, Cloning of the Gene That Encodes ACC Synthase Topic 22.5, Cloning of the Gene That Encodes ACC Oxidase Topic 22.6, Ethylene Binding to ETR1 and Seedling Response to Ethylene Topic 22.7, Conservation of Ethylene Signaling Components in Other Plant Species Topic 22.8, ACC Synthase Gene Expression and Biotechnology Topic 22.9, The hookless Mutation Alters the Pattern of Auxin Gene Expression Topic 22.10, Ethylene Inhibits the Formation of Nitrogen-Fixing Root Nodules in Legumes Topic 22.11, Ethylene Biosynthesis Can Be Blocked with Anti-Sense DNA Topic 22.12, Abscission and the Dawn of Agriculture Topic 22.13, Specific Inhibitors of Ethylene Biosynthesis Are Used Commercially to Preserve Cut Flowers Topic 22.14, Chapter Twenty-Two References 23. Abscisic Acid: A Seed Maturation and Stress-Response Hormone Topic 23.1, The Structure Of Lunularic Acid from Liverworts Topic 23.2, ABA May Be an Ancient Stress Signal Topic 23.3, Structural Requirements for Biological Activity of Abscisic Acid Topic 23.4, The Bioassay of ABA Topic 23.5, Evidence for Both Extracellular and Intracellular ABA Receptors Topic 23.6, The Existence of G Protein-Coupled ABA Receptors Is Still Unresolved Topic 23.7, The Yeast Two-Hybrid System Topic 23.8, Yellow Cameleon: A Noninvasive Tool for Measuring Intracellular Calcium Topic 23.9, Phosphatidic Acid May Stimulate Sphingosine-1-Phosphate Production Topic 23.10, The ABA Signal Transduction Pathway Includes Several Protein Kinases Topic 23.11, The ERA1 and ABH Genes Code for Negative Regulators of the The ABA Response Topic 23.12, Promoter Elements That Regulate ABA Induction of Gene Expression Topic 23.13, Regulatory Proteins Implicated in ABA-Stimulated Gene Transcription Topic 23.14, ABA Gene Expression Can Also Be Regulated by mRNA Processing and Stability Topic 23.15, ABA May Play a Role in Plant Pathogen Responses Topic 23.16, Proteins Required for Desiccation Tolerance Topic 23.17, The Types of Coat-Imposed Seed Dormancy Topic 23.18, Types of Seed Dormancy and the Roles of Environmental Factors Topic 23.19, The Longevity of Seeds Topic 23.20, Genetic Mapping Of Dormancy: Quantitative Trait Locus (QTL) Scoring of Vegetative Dormancy Combined with a Candidate Gene Approach Topic 23.21, ABA-Induced Senescence and Ethylene Topic 23.22, Chapter Twenty-Three References 25. The Control of Flowering Topic 25.1, Contrasting the Characteristics of Juvenile and Adult Phases of English Ivy (Hedera helix) and Maize (Zea mays) Topic 25.2, Regulation of Juvenility by the TEOPOD (TP) Genes in Maize Topic 25.3, Flowering of Juvenile Meristems Grafted to Adult Plants Topic 25.4, Characteristics of the Phase-Shifting Response in Circadian Rhythms Topic 25.5, Support for the Role of Blue-Light Regulation of Circadian Rhythms Topic 25.6, Genes That Control Flowering Time Topic 25.7, Regulation of Flowering in Canterbury Bells by Both Photoperiod and Vernalization Topic 25.8, The Self-Propagating Nature of the Floral Stimulus Topic 25.9, Examples of Floral Induction by Gibberellins in Plants with Different Environmental Requirements for Flowering Topic 25.10, The Effects of Two Different Gibberellins on Flowering (Spike Length) and Elongation (Stem Length) Topic 25.11, The Contrasting Effects of Phytochromes A and B on Flowering Topic 25.12, A Gene That Regulates the Floral Stimulus in Maize Topic 25.13, Chapter Twenty-Five References 26. Responses and Adaptations to Abiotic Stress Topic 26.1, Stomatal Conductance and Yields of Irrigated Crops Topic 26.2, Membrane Lipids and Low Temperatures Topic 26.3, Ice Formation in Higher-Plant Cells Topic 26.4, Water-Deficit-Regulated ABA Signaling and Stomatal Closure Topic 26.5, Genetic and Physiological Adaptations Required for Zinc Hyperaccumulation Topic 26.6, Cellular and Whole Plant Responses to Salinity Stress Topic 26.7, Signaling during Cold Acclimation Regulates Genes That Are Expressed in Response to Low Temperature and Enhances Freezing Tolerance Topic 26.8, Chapter Twenty-Six References

globalhealingcenter.com — The Benefits of Fruits & VeggiesVegetarians and CancerOther than lowering the risk of cancer, helping to control cholesterol and fat levels, fruits and vegetables provides loads of antioxidants, which help remove free radicals from the body.Free radicals may cause cellular damage and lead to cancer. Free-radical wrangling antioxidants are found in all fruits and vegetables, ... as well as raw nuts and seeds.Common antioxidants are vitamin A, vitamin E, vitamin C, selenium, lycopene, lutein and beta-carotene.Different fruits are higher in different types of antioxidants, so make sure you’re eating a wide variety...Looking for a quick and easy way to pack antioxidants into your diet? Bulk up on healthy berries! Not only do they contain extremely high amounts of antioxidants, they contain phytochemicals. ...Phytochemicals seem to block cancer development, but youll miss out on this important cancer fighting component if you just take an antioxidant supplement instead of consuming phytochemical containing foods, like berries.Blueberries are among the best free radical wranglers on the market, and they’re fun to gather at you-pick-farms.The American Cancer Society Guidelines states that 30-40% of the occurrences of cancer are linked with diet, and therefore preventable.You can decrease your cancer chances even more by eating a raw food diet of fruit, vegetables, nuts and seeds and drinking only water.

runningraw.com — What is the difference between Raw Food and Living Food? Although the definitions of raw vary, it is commonly held that for a food to be raw it must have not been heated over 118 degrees F. My personal belief is that foods begin to break down and lose nutritive value when subjected to temperatures over 100 degrees F. A living food may or may not be a raw food (it may have been cooked at one point), but it has been re-enlivened or populated with living cultures. Examples would be kombucha tea, miso, tempeh, kim chee and krauts, etc. What is a detox? Detox, short for detoxification, is the elimination of toxic substances from the body. What can I expect during my detox? The detox is a highly individual process. Everyone experiences it differently. For some there are no detox symptoms at all. My detox lasted 4 months. I was light-headed, nauseous, weak, tired, headaches, fever-like symptoms. It was not fun, but I came through the other side with a new body. Where do I get my protein? This is probably the most common question i get, and the answer is that I'm not really that concerned with protein intake. Yes, I do consume some protein in the few hemp seeds and nuts that I eat. The dark leafy greens and broccoli that I consume daily also contain protein, but all in all, I really don't consume that much protein. The human body breaks protein down into amino acids, so I cut out the middle man and eat foods that are rich in amino acids - ALL uncooked fruits and vegetables. How do I get enough calories? Actually, I consume much fewer calories than the average American... I'll be doing a caloric breakdown of a single day shortly... my guess is that my consumption falls short of 2000 calories. Raw food is a much more efficient fuel, whereas many of the calories consumed on a SAD diet are burned trying to break down the very food that's providing the energy, and to clean up the damage brought about by an unhealthy diet. Running Raw Diet How long have I been eating raw? I took the plunge into fantastic health on November 3rd of 2004. What do I eat on a daily basis? I don't really have a strict plan or routine when it comes to my daily consumption. I eat what feels good. On most mornings i'll start with a piece of fresh fruit or two (apple, bananas, orange, grapefruit, kiwi, peach, strawberries, etc...), then I'll have a Larabar sometime mid morning. Before my workouts I usually consume a banana and some young coconut water. After my workouts I'll have another piece or two of fresh fruit - within 15 minutes of completing my workout!!! Then when i get home I'll make a smoothie with fruit and greens (kale, lettuce, collard), a few dates and some dulse (for electrolytes). Mid evening I'll chomp on another Larabar, and then I'll make a massive salad at around 7pm... it's got tons of different greens, broccoli, red peppers, radishes, avocado, celery, snap peas, mushrooms and whatever else i can find to throw in there... every day is different... but this is somewhat normal for me, and gives me all the energy I need and more. Did I go vegetarian or vegan before going raw? I was vegan for 6 years before I went raw. The last six months before I went raw I was eating a macrobiotic vegan diet. What is my pre-race regimen? As for a pre-race dinner... I eat no later than 6pm the night of a race... and that meal is almost entirely fruit - bananas, apples, mangoes, kiwis, strawberries... just no melon (they don't play well with other fruit). I might also have a little romaine lettuce. Make sure you are very hydrated the day before the race. The morning of the race, I wake up 3 hours before my start time and have a large all fruit breakfast. Half an hour later I go for a 3 to 5 minute run to get my metabolism going (all the top runners do this). Then I relax and make sure I'm getting lots of fluids... I'll drink at least 32 ounces of water or coconut water before the race... I stop drinking 30 minutes before the start. What supplements do I take? Actually, I don't take any. The point of the Running Raw Project is to prove that one can accomplish incredible feats of physical health and performance using inexpensive, easy to find, fruits, vegetables, nuts and seeds. What superfoods do I take? My belief is that a raw lifestyle should be as sustainable and economically feasible as possible. Therefore, I keep to the foods that are commonplace in any supermarket anywhere in the country, and cost very little to purchase. The miracle of the raw diet is not in the foods you are consuming, it's in the foods you are NOT consuming. Your body is the miracle, you don't need expensive "superfoods" to have a super body. The Running Raw Project How did the project begin? The Running Raw Project came into existence on December 25th of 2005. I was at a Christmas party at my friend's house in Venice, CA. The topic of my recent entrance into the world of running had come up. As I described the changes that were happening to my body and the abnormal feats of endurance that I was capable of, someone said - "you should film this". That hadn't occured to me before. Had anyone ever done that? Was anyone documenting the physical changes that occur when one goes raw? Were people testing this diet and it's relation to physical performance? I looked around online and found not one reference to a Raw diet and athletic performance. This blew my mind. What I was experiencing was off the charts, was I the only one experiencing these physical improvements on this diet? I had to find out. Thus the journey began. What is the status of the documentary film? As of September of 2008, the documentary is on hold. Other aspects of the project have taken precedence. My hopes are that a new team will be assembled and a new and better film will be produced. Raw Food and Performance Coming Soon. Recipes My personal favorite organic smoothie recipe: 2 ripe bananas 6 large dates 3 large leaves of kale 2 tablespoons Nutiva hemp seeds 1 tablespoon flax seeds 1 tablespoon dulse flakes 1 dash cinnamon 2 cups filtered water Training What does my training regimen look like? It all depends on the type of event I'm training for and the time of year. Currently I'm running about 91 miles a week. Which is accomplished by two runs of 5 to 13 miles a day. I also incorporate leg strength and core strength routines 3 times a week. On Tuesdays I do two to four mountain ascents at just below race pace. Typically, the mountains run have between 900 to 1,500 foot vertical gain. Thursdays are reserved for speedwork on the track. The length and intensity of the intervals depends on the event that I'm training for. I compete in a race every weekend which serves as a tempo run. Each race is preceded by a 3 mile warmup and a minimum of a 3 mile warmdown. Did I start training right away when I went raw? I was raw for a year before I started to train. I don't think it's a good idea to be on a training regimen when you are starting a raw diet. The detox can be pretty intense, and the exercise can further the stress on your immune system. What is my resting heart rate? Resting heart rate is measured the moment you first wake up in the morning, or after a period of 20 minutes of no activity. Currently, my RHR is 38. Was I athletic before I went raw? I was a competitive Cross Country skier and track athlete in high school. I competed my freshman and part of my sophomore years in college, then "retired" at age 20. (Back To Top) Weight Loss Eating a raw or living foods diet is one of the most effective ways to safely lose weight and keep it off. It is not uncommon for people to lose 25 lbs or more their first month of going raw.